*, Satya Chintamanani2†, Ping He3, Hirotada Fukushige4, Liping Yu5, Meiyu Shao1, Lihuang Zhu5, David F. Hildebr"> A gain-of-function mutation in <i>Msl10</i> triggers cell death and wound-induced hyperaccumulation of jasmonic acid in <i>Arabidopsis</i>-文献传递-植物通论文库

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A gain-of-function mutation in Msl10 triggers cell death and wound-induced hyperaccumulation of jasmonic acid in Arabidopsis

作 者 ：Yan Zou1†*, Satya Chintamanani2†, Ping He3, Hirotada Fukushige4, Liping Yu5, Meiyu Shao1, Lihuang Zhu5, David F. Hildebr

Keywords:Arabidopsis, cell death, jasmonates, mechanosensitive ion channel, wound,

Abstract:

Jasmonates (JAs) are rapidly induced after wounding and act as key regulators for wound induced signaling pathway. However, what perceives the wound signal and how that triggers JA biosynthesis remains poorly understood. To identify components involved in Arabidopsis wound and JA signaling pathway, we screened for mutants with abnormal expression of a luciferase reporter, which is under the control of a wound-responsive promoter of an ethylene response factor (ERF) transcription factor gene, RAP2.6 (Related to APetala 2.6). The rea1 (R

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